Page 300 - The Vasculitides, Volume 1: General Considerations and Systemic Vasculitis
P. 300

274 Elizabeth J. Brant, Julie G. McGregor, J. Charles Jennette et al.

[43] Pendergraft WF III, Pressler BM, Jennette JC, et al. Autoantigen complementarity: a
       new theory implicating complementary proteins as initiators of autoimmune disease. J.
       Mol. Med. 2005; 83: 12-25.

[44] Kain R, Matsui K, Exner M, et al. A novel class of autoantigens of anti-neutrophil
       cytoplasmic antibodies in necrotizing and crescentic glomerulonephritis: the lysosomal
       membrane glycoprotein h-lamp-2 in neutrophil granulocytes and a related membrane
       protein in glomerular endothelial cells. J. Exp. Med. 1995; 181: 585-597.

[45] Kain R, Exner M, Brandes R, et al. Molecular mimicry in pauci-immune focal
       necrotizing glomerulonephritis. Nat. Med. 2008; 14: 1088-1096.

[46] Roth AJ, Brown MC, Smith RN, et al. Anti-LAMP-2 antibodies are not prevalent in
       patients with antineutrophil cytoplasmic autoantibody glomerulonephritis. J. Am. Soc.
       Nephrol. 2012; 23: 545-555.

[47] McGrath MM, Isakova T, Rennke HG, et al. Contaminated cocaine and antineutrophil
       cytoplasmic antibody-associated disease. Clin. J. Am. Soc. Nephrol. 2011; 6: 2799-
       2805.

[48] Pearson T, Bremmer M, Cohen J, et al. Vasculopathy related to cocaine adulterated
       with levamisole: A review of the literature. Dermatol. Online J. 2012; 18:1.

[49] Pendergraft WF III, Niles JL. Trojan horses: drug culprits associated with
       antineutrophil cytoplasmic autoantibody (ANCA) vasculitis. Curr. Opin. Rheumatol.
       2014; 26: 42-49.

[50] Lyons PA, Rayner TF, Trivedi S, et al. Genetically distinct subsets within ANCA-
       associated vasculitis. N. Engl. J. Med. 2012; 367: 214-223.

[51] Kamesh L, Heward JM, Williams JM, et al. CT60 and +49 polymorphisms of CTLA 4
       are associated with ANCA-positive small vessel vasculitis. Rheumatology (Oxford)
       2009; 48:1502-1505.

[52] Slot MC, Sokolowska MG, Savelkouls KG, et al. Immunoregulatory gene
       polymorphisms are associated with ANCA-related vasculitis. Clin. Immunol. 2008;
       128: 39-45.

[53] Lee YH, Choi SJ, Ji JD, et al. CTLA-4 and TNF-alpha promoter-308 A/G
       polymorphisms and ANCA-associated vasculitis susceptibility: a meta-analysis. Mol.
       Biol. Rep. 2012; 39: 319-326.

[54] Carr EJ, Niederer HA, Williams J, et al. Confirmation of the genetic association of
       CTLA4 and PTPN22 with ANCA-associated vasculitis. BMC Med. Genet. 2009; 10:
       121.

[55] Tsuchiya N, Kobayashi S, Kawasaki A, et al. Genetic background of Japanese patients
       with antineutrophil cytoplasmic antibody-associated vasculitis: association of HLA-
       DRB1*0901 with microscopic polyangiitis. J. Rheumatol. 2003; 30: 1534-1540.

[56] Jagiello P, Aries P, Arning L, et al. The PTPN22 620W allele is a risk factor for
       Wegener's granulomatosis. Arthritis. Rheum. 2005; 52: 4039-4043.

[57] Cao Y, Schmitz JL, Yang J, et al. DRB1*15 Allele Is a Risk Factor for PR3-ANCA
       Disease in African Americans. J. Am. Soc. Nephrol. 2011; 22: 1161-1167.

[58] Luo H, Chen M, Yang R, et al. The association of HLA-DRB1 alleles with
       antineutrophil cytoplasmic antibody-associated systemic vasculitis in Chinese patients.
       Hum. Immunol. 2011; 72:422-425.

[59] Jennette JC, Falk RJ. The pathology of vasculitis involving the kidney. Am. J. Kidney
       Dis. 1994; 24: 130-141.

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