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220 Loic Guillevin
[7] Scott DG, Bacon PA, Elliott PJ, et al. Systemic vasculitis in a district general hospital
1972–1980: clinical and laboratory features, classification and prognosis of 80 cases. Q.
J. Med. 1982; 51:292-311.
[8] McMahon BJ, Heyward WL, Templin DW, et al. Hepatitis B-associated polyarteritis
nodosa in Alaskan Eskimos: clinical and epidemiologic features and long-term follow-
up. Hepatology 1989; 9:97-101.
[9] Reinhold-Keller E, Herlyn K, et al. No difference in the incidences of vasculitides
between north and south Germany: first results of the German Vasculitis Register.
Rheumatology 2002; 41:540-549.
[10] Watts RA, Gonzalez-Gay MA, Lane SE, et al. Geoepidemiology of systemic vasculitis:
comparison of the incidence in two regions of Europe. Ann. Rheum. Dis. 2001; 60:170-
172.
[11] Watts RA, Lane SE, Scott DG, et al. Epidemiology of vasculitis in Europe. Ann.
Rheum. Dis. 2001; 60:1156-1157.
[12] Mahr A, Guillevin L, Poissonnet M, et al. Prevalences of polyarteritis nodosa,
microscopic polyangiitis, Wegener's granulomatosis, and Churg–Strauss syndrome in a
French urban multiethnic population in 2000: a capture–recapture estimate. Arthritis.
Rheum. 2004; 51:92-99.
[13] Trepo CG, Zucherman AJ, Bird RC, et al. The role of circulating hepatitis B
antigen/antibody immune complexes in the pathogenesis of vascular and hepatic
manifestations in polyarteritis nodosa. J. Clin. Pathol. 1974; 27:863-868.
[14] Guillevin L, Ronco P, Verroust P. Circulating immune complexes in systemic
necrotizing vasculitis of the polyarteritis nodosa group. Comparison of HBV-related
polyarteritis nodosa and Churg Strauss angiitis. J. Autoimmun. 1990; 3:789-792.
[15] Fye KH, Becker MJ, Theofilopoulos AN, et al. Immune complexes in hepatitis B
antigen-associated periarteritis nodosa. Detection by antibody-dependent cell-mediated
cytotoxicity and the Raji cell assay. Am. J. Med. 1977; 62:783-791.
[16] Trepo C, Thivolet J. Hepatitis associated antigen and periarteritis nodosa (PAN). Vox.
Sang. 1970; 19:410-411.
[17] Prince AM, Trepo C. Role of immune complexes involving SH antigen in pathogenesis
of chronic active hepatitis and polyarteritis nodosa. Lancet 1971; 1:1309-1312.
[18] Mason A, Theal J, Bain V, et al. Hepatitis B virus replication in damaged endothelial
tissues of patients with extrahepatic disease. Am. J. Gastroenterol. 2005; 100:972-976.
[19] Cid MC. New developments in the pathogenesis of systemic vasculitis. Curr. Opin.
Rheumatol. 1996; 8:1-11.
[20] Le Tonqueze M, Dueymes M, Giovangrandi Y, et al. . The relationship of anti-
endothelial cell antibodies to anti-phospholipid antibodies in patients with giant cell
arteritis and/or polymyalgia rheumatica. Autoimmunity 1995; 20:59-66.
[21] Jamin C, Dugue C, Alard JE, et al. Induction of endothelial cell apoptosis by the
binding of anti-endothelial cell antibodies to Hsp60 in vasculitis-associated systemic
autoimmune diseases. Arthritis. Rheum. 2005; 52:4028-4038.
[22] Grau GE, Roux-Lombard P, Gysler C, et al. Serum cytokine changes in systemic
vasculitis. Immunology 1989; 68:196-198.
[23] Panegyres PK, Blumbergs PC, Leong AS, et al. Vasculitis of peripheral nerve and
skeletal muscle: clinicopathological correlation and immunopathic mechanisms. J.
Neurol. Sci. 1990; 100:193-202.
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